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The release of Cu into tumor cells enhances reactive oxygen species (ROS) as well as nitric oxide (NO) production, which results in DNA fragmentation, induces pro-apoptotic protein upregulation such as Bax, and enhances caspase-3, caspase-8, and caspase-9 generation that motivates cancer cells apoptosis [68] even in solid tumor hypoxic condition [69]

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Expert Opin Investig Drugs 28(2):191205 van Opdenbosch N, Lamkanfi M (2019) Caspases in cell death, inflammation, and disease [J]

(2020) demonstrated protective effects of GHK-Cu in a bleomycin-induced pulmonary fibrosis model, showing anti-oxidative stress and anti-inflammatory activity in lung tissue

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